By Vishal Madan
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McLaren, K. & Benton, E. (1997). Prevailing papillomavirus types in non-melanoma carcinomas of the skin in renal allograft recipients. A. & Proby, C. (2002). Human papillomaviruses and non-melanoma skin cancer. ; Schmook, T. & Meyer, T. (2004). Human papillomaviruses in transplant-associated skin cancers. ; Pukkala, E. & Karvonen, J. (1999). Basal cell skin carcinoma and other nonmelanoma skin cancers in Finland from 1956 through 1995. S. (1999). The mysteries of geographic variability in nonmelanoma skin cancer incidence.
4. Comparison of expression of CD44v6 (a), E48 (b) and U36 (c) in small frozen sections of BCCs, magnification 200x. 3 Markers p53 and Ki-67 in BCCs and BCC-free tumor margin tissue Molecular alterations can be found during carcinogenesis and tumor growth. As illustrated schematically in Figure 1 (cf. 1), the inactivation of tumor suppressor gene p53 is an important step towards carcinogenesis. During inactivation and mutation of p53 mutated p53 protein is formed. Mutated and wildtype p53 protein can be detected immunohistochemically by different or identical antibodies, for instance in tissues with field cancerization after intensive solar irradiation (60, 62) as well as in tumor-free margin surrounding of BCCs (98, 157 to 161).