Cancer a Comprehensive Treatise 2: Etiology: Viral by J. Michael Bishop, Harold E. Varmus (auth.), Frederick F.

April 3, 2017 | Cancer | By admin | 0 Comments

By J. Michael Bishop, Harold E. Varmus (auth.), Frederick F. Becker (eds.)

The effect of uncomplicated study on oncology has been fairly remarkable within the contemporary look for the reason for malignancy. both notable is our appreciation of the reason for tumors in keeping with statement. Even within the earliest period of the research of infectious illnesses, it was once proposed that tumorous development in animals and birds resulted from "minute" infectious debris. Experiments then supported the speculation, that the etiologic agent in lots of animal tumors used to be viral. the advance of molecular biology, supported via technical advances and conceptual knowing of macromolecular motion, ended in an explosive elevate in reports of animal oncogenic viruses. For a decade, new findings emerged from learn laboratories revealing the big number of such brokers, the advanced­ ity in their interactions with cells, and the tantalizingly attainable mechanisms wherein they may reason malignant transformation of the mobile. again and again, clues emerged which instructed the intervention of viral brokers in human tumors. A breathless pleasure pervaded either the medical and public groups as hugely publicized findings speedily each other. the thrill used to be no much less medical than it was once sensible, for implicit within the idea of the viral oncogen is the opportunity of particular virostatic or virotoxic brokers or of immunization.

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1966, Metabolic requirements for infection by Rous sarcoma virus. 1. Transient requirement for DNA synthesis, Virology 29:444. BADER, J. , 1972a, Metabolic requirements for infection by Rous sarcoma virus. IV. Virus reproduction and cellular transformation without cellular division, Virology 48:494. BADER, J. , 1972b, Temperature-dependent transformation of cells infected with a mutant of Bryan Rous sarcoma virus,]. Virol. 10:267. BADER, J. , 1973, Virus-induced transformation without cell division, Science 180: 1069.

3. Density of Cells at Saturation on a Solid Surface Under defined conditions of growth, transformed cells can grow to a greater number on a given surface area than can normal cells. This is a controversial and tenuous criterion, useful only if conditions of growth are carefully controlled. The mechanisms which limit cellular multiplication are not known, but the once popular concept of inhibition due to contacts among cells has fallen into disrepute. Present opinion holds that "there is no experimental support for the concept that 'contact inhibition' ...

Human tumors are induced by infection with RNA tumor viruses which do not replicate in human tissue: man is a non permissive heterologous host for RN A tumor viruses derived from other species. According to this scheme, virus particles will not be available from tumor tissue for characterization and preparation. However, virus-specific DNA and RNA, and possibly viral antigens, should be present in tumors. , homologous) reagents are available. This necessitates a survey with materials from available animal viruses in the hope that one can identify either the etiological agent or a related virus.

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